Tetracaine HCl - Essential Information:

High-Purity Pharmaceutical Anesthetic HCl Powder CAS 136-47-0 - Tetracaine & Lidocaine
Lidocaine hydrochloride undergoes rapid metabolism in the liver, with both metabolites and unchanged drug being excreted via the kidneys. Key biotransformation processes include oxidative N-dealkylation, ring hydroxylation, amide linkage cleavage, and conjugation. The primary pathway, N-dealkylation, produces metabolites monoethylglycinexylidide and glycinexylidide, which exhibit similar but less potent pharmacological and toxicological effects as Lidocaine hydrochloride. Approximately 90% of administered Lidocaine hydrochloride is excreted in the form of various metabolites, with less than 10% being excreted unchanged. The main urinary metabolite is a conjugate of 4-hydroxy-2,6-dimethylaniline.

Lidocaine HCl: A Premium Amide Local Anesthetic.
As the dosage increases, both the efficacy and toxicity rise. At sub-poisoning plasma concentrations, Lidocaine HCl exhibits an anti-convulsive effect.
Blood concentrations exceeding 5μg/ml may induce convulsions.

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