Tetracaine HCl Essentials:

Pharmaceutical Anesthetic Purity HCl Powder CAS 136-47-0 - Tetracaine Lidocaine
Lidocaine hydrochloride is swiftly processed by the liver, with both its metabolites and the unchanged drug being excreted via the kidneys. The biotransformation journey involves pivotal actions such as oxidative N-dealkylation, ring hydroxylation, amide linkage cleavage, and conjugation. A major biotransformation pathway, N-dealkylation, produces metabolites like monoethylglycinexylidide and glycinexylidide, whose pharmacological and toxicological effects mirror those of Lidocaine hydrochloride, albeit with reduced potency. Astonishingly, around 90% of administered Lidocaine hydrochloride is excreted as diverse metabolites, leaving less than 10% in its unchanged form. The urine's primary metabolite is a conjugate of 4-hydroxy-2,6-dimethylaniline.

Lidocaine HCl stands as a premier amide local anesthetic.
With increased dosage comes heightened effects or toxicity, showing an anti-convulsive impact at plasma concentrations below toxic levels.
Blood concentrations exceeding 5μg ? ml-1 may induce convulsions.

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